Biophoton Emission In Humans

 


A particularly intriguing presentation of biophotons in association with humans cannot be missed: The biophoton intensity seems to be modulated by intention or human interaction during healing or other various kinds of bioenergy practices. Links have also been suggested between biophotons and observations that are difficult to measure and quantify, including consciousness, mind–matter manipulation, and paranormal phenomena at the moment of death. One could argue that an experimental control that appears common to all these phenomena is the involvement of human factors, which at the physical level will reside in the engagement of neuronal activities, no matter whether autonomic or intentional.

The variation in surface biophoton emission has been known to correlate with changes in metabolism. When living organisms become stressed, the ROS varies to subsequently change biophoton emission. Induced changes in photon emissions offer physiologically relevant explanations for some human experiences that are often inexplicable even though not uncommon, including the entopic phenomena of phosphene and negative afterimage. Studies have also suggested that ROS may be signaling agents, rather than simply potential toxic agents. Whether ROS are signaling or toxic agents, the variation of the rate of the production of ROS in humans will be regulated ultimately by autonomic neuromodulation, which will likely differ at different stages of life and also be perturbed or affected by environmental factors and acquired conditions. Variations in biophoton emissions due to variations of ROS then shall be expected to follow the temporal paces as well as spatial patterns of the neuromodulating perturbation causing autonomic adaptation of ROS production, with a potential delay in the phase of change.      

Biophoton emission remains controversial. The photo-genic origin of biophoton has been attributed to the oxidative stress or free radical production. However, there are considerable gaps in quantitative understanding of biophoton emission. Dr. Daqing Piao proposes an analytical hypothesis for interpreting a few patterns of steady-state biophoton emission of human, including the dependency on age, the diurnal variation, and the geometric asymmetry associated with serious asymmetrical pathological conditions. The hypothesis is based on an alternative form of energy state, termed vivo-nergy, which is associated with only metabolically active organisms that are also under neuronal control. The hypothesis projects a decrease of the vivo-nergy in human during growth beyond puberty. The hypothesis also proposes a modification of the vivo-nergy by the phases of systematic or homeostatic physiology. The hypothesis further postulates that the deviation of the physiology-modified vivo-nergy from the pre-puberty level is deteriorated by acquired organ-specific pathological conditions. A temporal differential change of vivo-nergy is hypothesized to proportionally modulate oxidative stress that functions as the physical source of biophoton emission. The resulted steady-state diffusion of the photon emitted from a photo-genic source in a human geometry simplified as a homogeneous spherical domain is modeled by photon diffusion principles incorporating an extrapolated zero-boundary condition. The age and systematic physiology combined determines the intensity of the centered physiological steady-state photo-genic source. An acquired pathology sets both the intensity and the off-center position of the pathological steady-state photo-genic source. When the age-commemorated, physiology-commanded, and pathology-controlled modifications of the steady-state photo-genetic sources are implemented in the photon diffusion model, the photon fluence rate at the surface of the human-representing spherical domain reveals the patterns on age, the temporal variation corresponding to systematic physiology, and the geometric asymmetry associated with significant asymmetric pathological condition as reported for spontaneous biophoton emission. The hypothesis, as it provides conveniences for quantitative estimation of biophoton emission patterns, will be extended in future works towards interpreting the temporal characteristics of biophoton emission under stimulation.

Dr. Daqing Piao received a BS degree in 1990 in physics from Tsinghua University and earned MS and PHD degrees in 2001 and 2003, respectively, both in biomedical engineering within the Electrical and Computer Engineering program from University of Connecticut. He is now a Professor in the School of Electrical and Computer Engineering at Oklahoma State University. His research interests include light-tissue interaction and mind-matter-interaction.

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